Dr Simeons Describes How the HCG Hormone Effects Fat Cells Around the Belly
Dr Simeons HCG diet depends on how HCG assists in mobilizing fat cells from around the abdomen and trunk as opposed to fat deposited on the buttocks, arms, legs. Why is this so important from a medical point of view?
Dr Simeons explains these fats and weight loss: “When an obese patient tries to reduce by starving himself, he will first lose his normal fat reserves. When these are exhausted he begins to burn up structural fat, and only as a last resort will the body yield its abnormal reserves, though by that time the patient usually feels so weak and hungry that the diet is abandoned. It is just for this reason that obese patients complain that when they diet they lose the wrong fat. They feel famished “
Dangers of Fat Around Belly and Buttocks
Fat tissue accumulated preferentially around the belly (apple patter) and buttock (pear pattern) has more adverse effects on health than does fat stored in arms and legs.
Fat storage is easily measured by waist circumference:
- In women, the normal waist circumference should be less than 36 inches
- In men about 38-40 inches.Above these values increasing fat deposits are associated with greater risks for diabetes, hypertension and heart disease. Some of the cardiovascular risk may be due to the increase in abnormalities of blood vessels including development of plaques and thickening of the blood vessel wall.
What Determines Fat Deposition?
Steroids, growth and sex hormones, genetic factors, and physical activity are all determinants of body composition In addition, body composition and large artery properties may be influenced by behavioral characteristics such as smoking,and alcohol consumption.
Differences in metabolism of fat tissue
Fat metabolism varies depending on whether it is located centrally in the trunk or in the limbs Fat cells from trunk regions are more sensitive todestruction(lypolysis.) The higher lipolytic activity of the fat cells in the trunk leads to overexposure of the liver to free fatty acids resulting in insulin resistance and hyperinsulinemia. In addition, fat cells are endocrine organs that produce many peptides, and chemicals with wide spread effect on many organs. Regional differences in secretion of these peptides could thus potentially explain the direct and opposite effects of these fat depots on arterial properties.
Short Reports on Experimental Evidence of HCG Effecting Fat Cells from the Medical Literature
Throughout the years, hCG has been reported to exert its actions on several tissues other than gonadal.Therefore, we are not dealing with a “pure” sex hormone: Several clinical conditions, such as asthma, allergies, gastric ulcers, intermittent claudication of the lower limbs and anemia were treated with hCG, with encouraging results. Available data indicate that hCG might also improve lypolisis(fat cell destruction) in human adipose tissue, via an inhibitory effect on lipogenesis.(new fat cell production.)
Fleigelman “concluded that the administration of hCG in rats decreased the activity of alfa-glycerophosphate dehydrogenase and glucose-6-phosphate dehydrogenase from the liver and adipose tissue, suggesting a decreased lipogenic activity in both tissues under hCG.”
Yanagihara reported that hCG accelerates “not only the mobilization of fat from fat deposits, but also its utilization in peripheral tissues. hCG increased the metabolism of injected fat emulsions, suggesting the acceleration not only of fat oxidation, but also increased ketone production in the liver and its utilization in peripheral tissues.”
Romer reported that “hCG intensifies the metabolism of rat brown adipose tissue. Administration of hCG to humans appears to increase the release of fatty acids that varies with the age of the subject”
. Melichar demonstrated that “hCG causes a marked FFA release in newborn infants.In adults, a single dose of hCG caused a marked FFA release by when compared to placebo-treated subjects.”
The last author attributes some of the report weight loss to this effect on fatty acid release and eventual metabolism.